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6.1 Mechanisms underlying treatment of UC The treatment of UC involves the modulation of various immunoinflammatory pathways, including the activation and suppression of key mechanisms such as PPAR, Nrf2, NF-kB, and macrophages, along with the regulation of the Th17/Treg balance [61]

Wang, X., Wu, N., Sun, C., Jin, D

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Similarly, DSePA, another interesting organoselenium compound, has shown potent efficacy as a lung-specific radiomitigator through inducing the expressions rather than mimicking the activities of seleno-GPxs, thus warranting future clinical studies [61, 62]