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Therefore, in this review we detail the direct and indirect molecular mechanisms by which incretin receptor agonists inhibit fibrotic signalling pathways, and critically appraise the evidence for their preclinical and clinical efficacy in fibrotic disorders across diverse tissues, including the liver, heart, kidney, lung, and joints (Table 1

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it reflects overlapping curiosity about three compounds that happen to be studied within the same broader field

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PAF-acetylhydrolase (PAF-AH) is an extracellular phospholipase A2 which hydrolyzes