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however, several hypotheses informed largely by evidence from related GLP-1 receptor agonist classes have been proposed: Weight loss-mediated reduction in vascular resistance , as excess adiposity is a well-established driver of hypertension

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Sleep architecture disruption

Endogenous GLP-1 released from the gut acts directly on vagal afferents, which relay information from the abdomen to the brain and regulate multiple physiological systems

IKK phosphorylates targets in the NF-B signaling pathway, thereby activating the NF-B pathway