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It has some research for autoimmune conditions and immune deficiency, but as a general "immune optimizer"

In 1993, it was discovered that Wilsons disease was caused by a mutation in the ATP7B gene, on chromosome 13, which resulted in absent or reduced function of a copperchaperone protein, ATP7B.14 ATP7B is a metal-transporting P-type ATPase, located on the trans-Golgi complex of the hepatocyte.15 The ATP7B protein is necessary for transport of copper into vesicles that form lysosomes for excretion into the bile

Nutr Res Rev 23, 200229.Google Scholar Schll, M, Verberk, IMW, del Campo, M et al

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