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Hyperhomocysteinemia is recognized as an independent risk factor for artherosclerosis of the coronary, cerebral, and peripheral vasculature

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Therefore, our results indicate that cholesterol levels regulate the viability of neuronal cells exposed to A, inducing pyroptosis, a proinflammatory death, which is counteracted by the recovery of mitochondrial GSH levels and the consequent reduction of A-induced oxidative stress

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