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General mitochondrial trifunctional protein (TFP) deficiency as a result of either alpha- or beta-subunit mutations exhibits similar phenotypes because mutations in either subunit alter TFP complex expression and subunit turnover

The commenter recommended that CMS propose an improvement activity that better reflects the expected roles and workflows of anesthesiologists

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While ongoing research underscores its potential as a breakthrough in regenerative therapy, further human trials are essential to fully elucidate its long-term safety, efficacy, and clinical applications.

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