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These in vivo results align with the in vitro data, consolidating the understanding that GHK-Cu exerts its protective effects in ALI by inhibiting excessive inflammatory responses through its anti-inflammatory and antioxidant properties, mediated by the suppression of NF-B and p38 MAPK signaling pathways [1]

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These benefits extend to tissue remodeling and improving heart function after myocardial infarction, highlighting IGF-1s potential in supporting overall health [2]

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