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Conclusion This study clarifies the key role of dopamine and D2 receptors in the mOFC in chronic migraine: in CM model mice, dopamine release in the mOFC is increased, which activates dopamine D2 receptor and inhibits excitatory DRD2+ neurons dominated by Camk2a, thereby improving pain hypersensitivity and anxiety-like behavior

[5] GURNEY, T., BROUNER, J., SPENDIFF, O

These differences may have resulted in a relative reduction in effect sizes when pooling cross-sectional estimates obtained in the main analyses and those from 6-year follow-up analyses

And when someone eventually stops taking the drug, the weight often returns

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