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Oxidative stress may then damage mitochondrial membranes and enzymes, further lowering ATP production and increasing the need for glutathione, SOD, catalase and other antioxidant defenses

The authors described these structural models as the basis for rational development of p53 inhibitors targeting the senescent cell biology field and diseases linked to cellular senescence including cancer

144146 Delphinidin exhibited anti-proliferative and pro-apoptotic effects in a dose-dependent manner on human endometriosis-derived cell lines by regulating the cell cycle according to the results of Park S

During radiotherapy, depleting GSH improves treatment efficacy by impairing the cells ability to neutralize ROS [85]