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Figure 1 There are some limitations in this review, as we did not include clinical trials or pay close attention to potentially protective mechanisms of agents in the pathophysiology of animal stroke
This study is part of a trial that started 42 d prior to expected calving and lasted until d 128 postpartum (pp)
However, the Glu 138 -Phe 143 sequence clearly has -helical propensity, which is in agreement with the recent crystal structure of the homologous glucagon receptor TM domain showing an extracellular -helical extension of TM1 20
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