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Beyond blood sugar, glycine exhibits anti-inflammatory effects by downregulating proinflammatory cytokines such as TNF- and IL-6, which are often elevated in metabolic syndrome and age-related chronic inflammation

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This could also be explained, in part by the fact that luminal A breast cancer cells, unlike our model of TNBC, are lacking expression of CD44, which has been shown to be the main effector of iron uptake, a key mechanism in the induction of ferroptosis [23, 43]

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