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Iron redox activity, while integral to numerous physiological processes, can catalyze ROS formation and lipid peroxidation, directly linking iron dysregulation to CHD development (15)

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NAD(P)H, one of the principal reductants, is produced by the pentose phosphate pathway (PPP) and can be phosphorylated by NAD kinase (NADK) to synthesize NADPH, which inhibits peroxidation damage caused by ferroptosis