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Specifically, mitochondrial complex I is believed to be the main component of mitochondria affected in PD as Complex I of the electron transport chain is left vulnerable to oxidative damage (Vali et al., 2007)

Acute JNK activation can be pro-apoptotic (e.g., in response to TNF- or oxidative stress), but it also plays essential roles in cell survival, metabolism, and tissue repair depending on the duration of activation and the cellular environment (70)

Data availability Sequencing data are available by contacting the corresponding author on reasonable request

Scientists found that glutathione supplements reversed H2O2-induced cytotoxicity and ROS generation by a lot in cell studies