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The scheme of Figure 2 describes a chemical-biological mechanism for the generation of intracellular oxidative stress that is caused by a continuous depletion of the cellular reduced glutathione pool by catalytic doses of Mercury
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Sempos CT
Instead, GHK-Cu appeared to act as a broad-spectrum homeostatic regulator, capable of resetting gene expression patterns in cultured cells towards those characteristic of a younger, more regenerative state
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