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Concurrently, accumulating evidence demonstrates that Ser637 phosphorylation participates in circadian regulation of mitochondrial fission (Serasinghe and Chipuk, 2017), highlighting the need for systematic elucidation of its spatiotemporal regulatory mechanisms and precise molecular determinants governing mitochondrial fission dynamics

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The active-site cysteine residue of Ca(2+)/calmodulin-dependent protein kinase I is protected from irreversible modification via generation of polysulfidation

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