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Collectively, these data reveal that exendin 2029 acts as a key sequence in inducing analgesia in CAP-elicited nociceptive behaviors in mice without causing hyperthermia, and this analgesic effect is mediated by exendin 2029 directly inhibiting CAP-induced TRPV1 activation but not proton-induced TRPV1 activation

The conditions used were as follows: Product ion scan in positive mode, precursor m/z 271 for APAP-CYS and 229 for PAP-CYS, curtain gas 25 psi, collision gas medium, ion spray voltage 3000 V, temperature ambient, ion source gases 16 psi/0 psi, entrance potential 10 V, declustering potential 60 V, collision cell exit potential 10 V, collision energy 25 V

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It is relevant for studies involving GLP-1 signaling, insulin-related pathways, appetite regulation, and gastric emptying research