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Therefore, we hypothesize that deregulated homocysteine metabolism in homocystinuria with severe hyperhomocysteinemia will lead to the cyclization of homocysteine into homocysteine thiolactone, which may render LDL less susceptible to oxidation

However, it also introduces additional complexity and potential side effects, as the dysesthesia signal demonstrates

(2024).Available from: [Google Scholar] 28.Structure Therapeutics Announces Positive Results from Phase 1b Clinical Study of Oral GLP-1 Receptor Agonist GSBR-1290 and Provides Program Update | Structure Therapeutics

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