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If side effects are problematic, we adjust extending the time at a dose, adding supportive measures, or exploring a medication switch

1, 5 July 2024, article 629

But even as access expands dramatically, new global evidence is raising concerns about the risks associated with discontinuing these therapies casting a shadow over what otherwise appears to be a watershed moment for patients

Direct binding of eNOS to the scaffolding domain of Cav-1 is a well-accepted mechanism to repress eNOS activity that is associated with endothelial dysfunction and cardiovascular disease 39,40