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A common pathological mechanism of co-morbidity is a prerequisite for the administration of the same drug to achieve common remission

PYC treatment reduces ROS production in HCV replicon cell lines, thereby limiting viral replication

CDI triggers metabolic shifts that could facilitate steatosis and inflammation, suggesting that CDI could be a risk factor for metabolic liver diseases

The inability to appropriately sense, buffer, or correct proteotoxic stress results in the accumulation of misfolded proteins, organelle dysfunction, aberrant signaling, and ultimately tissue failure

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