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HyDKA patients had poorer glycemic control and lower C-peptide than euDKA patients

In research, its primarily studied for its ability to stimulate the pituitary gland to release growth hormone

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277 circArhgap5-2 silencing results in inhibited lipid accumulation and adipose marker (PPAR, AdipoQ Cebp, and FABP4) downregulation

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