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But they work through different pathways, target different outcomes, and suit different patient profiles

There is overwhelming evidence that demonstrates that elevated calcium concentration, inflammation, increased oxidative stress, as well as a reduction in blood flow, are some of the pathophysiological mechanisms underlying NIHL (Figure 1

Suppression of cytosolic NADPH pool by thionicotinamide increases oxidative stress and synergizes with chemotherapy

PMID: 21875347

A photo-crosslinked gelatine methacryloyl (GelMA) loaded with puerarin (Pue) inhibits neutrophil and eosinophil aggregation while promoting matrix remodelling through the TGF-/MMP pathway in a rabbit POP model (Qin et al