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Despite the inbred nature of NOD mice, substantial inter-individual variability was observed, reflecting differences in disease kinetics and therapeutic response and mirroring clinical heterogeneity in high-risk populations, and underscoring the need for personalised approaches [67]

they were designated practice 1 (7,502 patients), practice 2 (5,299 patients) and practice 3 (6,470 patients)

Retrieved from Nollet, M., Wisden, W., Franks, N

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Together, these results suggest that CB0313.1 may improve host glucose homeostasis via the GLP-1/insulin/gluconeogenesis pathways