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NO modulates mitochondrial biogenesis through different routes, a selective one is by increasing the phosphorylated-active form of AMP-activated protein kinase (AMPK) that in turn induces PGC-1a signaling pathway [72]

CJC 1295 and BPC 157 : These peptides have boosted muscle and improved healing in animal studies, but the FDA recently banned them due to a lack of human research regarding their safety (11)

This could explain why infection of mice with oral Trepomnema denticola bacteria induces Tau hyperphosphorylation and A accumulation in mice in the hippocampus with neuronal loss (Wu et al., 2022), and why filtrates of Helicobacter pylori bacteria induce Tau hyperphosphorylation in mouse neuroblastoma cells and rat brains via GSK-3 activation (Wang et al., 2014)

Overall, the PubMed search showed that the use of the glutathione transporter as a target for developing brain-targeting agents was a relatively new and less explored field