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After receiving a complete description of the study, all participants provided informed written consent (ClinicalTrials.gov registration: NCT04977674)

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They have observed that glutamate receptor activation activated NOS, leading to an increased Ca 2+ influx through NMDA-controlled channels via NO synthesis (Besancon et al

15 Collectively, these findings suggest that GABA dysregulation contributes to impaired cortical connectivity and fear extinction deficits, 10,14 suggesting GABA is a potential therapeutic target for PTSD intervention

Gonzlez-Molina V, Mart-Pineda A, Gonzlez N