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Malkov A, Popova I, Ivanov A, Jang S-S, Yoon SY, Osypov A, Huang Y, Zilberter Y, Zilberter M (2021) A initiates brain hypometabolism, network dysfunction and behavioral abnormalities via NOX2-induced oxidative stress in mice

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aeruginosa ( Figure 6 ) and viability by scavenging ROS and free radicals directly (electron donation through the thiol moiety) or indirectly via GRed (this study) and catalase activity ( In addition to oxidative stress cytotoxicity, pyocyanin pathogenicity has shown to alter calcium homeostasis and depleting intracellular cAMP and ATP levels ( The effect of GSH in neutralizing epithelial cellular cyto-toxicity has been investigated in this study