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In conclusion, the present study demonstrated that semaglutide promoted the proliferation and osteogenic differentiation of BMSCs through the Wnt/LRP5/-catenin signaling pathway, and proved that LRP5 may be a key factor in GLP-1 promoting osteogenesis, which has certain positive implications for improving BMSCs function in OP patients with T2DM

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In addition, GLP-1RAs inhibit superoxide anion formation and ensuing VSMC senescence through suppressing Rac1 activation via cAMP/PKA pathways [123]